Roles of Nitricoxide Signaling Pathway in Atherosclerosis
Roles of Nitricoxide Signaling Pathway in Atherosclerosis
Atherosclerosis: Open Access (Omics Publishing S.L) has announced almost 50 percent discount on article processing charge to commemorate its 10th Anniversary.
Atherosclerosis: Open Access is an open access, peer-reviewed journal that focuses and welcomes submissions on all aspects of Coronary Heart disease, Stroke, Peripheral Arterial Disease, Chronic Kidney Disease, arteriosclerosis, Coronary Atherosclerosis, intracranial atherosclerosis etc.
It gives us great pleasure to announce the call for paper on the occasion of 10th Anniversary of the Journal at special and hefty discount of up to 50 percent on one-time article processing charge. Prospective academicians and scientists are encouraged to utilize this opportunity to get their articles reviewed, processed and published at relatively faster pace and at lower charges. In addition to this, the authors who publish with us during the year-long celebrations will also be eligible for academic awards recommended by the editorial panel.
The Archive page contains wide variety of articles such as Research / Review / Case reports / short communication / Mini review / Prospective / Letter to Editors Etc. We would like introduce a Review article which has been spread to the widest audience of experts; and thus increased in readership, citations and altimetry score.
Title: “Roles of Nitricoxide Signaling Pathway in Atherosclerosis”
Abstract: Nitric oxide (NO) generated by endothelial nitric oxide synthase (eNOS) and neuronal nitric oxide synthase(nNOS) plays a critical role in the vasoprotective function of the endothelium under physiological conditions. However, under pathological conditions, eNOS and nNOS become dysfunctional, and inducible nitric oxide synthase (iNOS) is stimulated to produce excessive NO, which induce endothelial dysfunction. NO signaling pathways mainly include phosphatidylinositol 3-kinase (PI3K)/serine threonine protein kinase B(AKT)/eNOS pathway, nuclear factorkappa B(NF-κB)/iNOS pathway, extracellular regulated protein(ERK)1/2/nNOS pathway, etc. which are involved in the development and progression of atherosclerosis through affecting NO synthesis and its functions. Meanwhile, NO-soluble guanylyl cyclase (sGC)-cyclic guanosine monophosphate (cGMP) pathway is closely related to atherosclerosis, due to the ability of regulating the degree of vasodilatation. Elucidating NO signaling pathway associated with atherosclerosis is necessary for the improvement and treatment of atherosclerosis. This review summarized the relationships between the different NO signaling pathways and atherosclerosis in the present relative studies.
To have a glance at the full length manuscript, you can visit us at our archive page and Currently, Journal’s Archive is holding not only normal issues but also focusing on special issues. The purpose of special issues is to publish the most exciting research with respect to the subjects of Atherosclerosis / Cardiovascular Diseases and to provide a rapid turn-around time regarding reviewing and publishing, and to publicize the articles freely for research, teaching and reference purposes. Submit manuscript of your research articles or special issue articles online through manuscript submission or forwarded to the Editorial Office at atherosclerosis@oajournal.org
Regards,
Steven Edwards
Managing Editor
Atherosclerosis: Open Access